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    It was found that inhibition of PDE-4 isoenzyme can elicit some anti-inflammatory diazepam effects [39] .
    The effect of diazepam on phosphodiesterases especially PDE-4 could be also part of the mechanisms involved in some experimental and clinical diazepam effects of benzodiazepines which are not well understood .
    Interestingly, diazepam has some effects such diazepam as airway smooth muscle relaxation [40] and hyperglycemia which are shared by PDE-4 inhibitors [41] and also, it is known that diazepam inhibits antigen-induced eosinophil infiltration into guinea-pig conjunctiva [42] as well as, other immune cell functions such as phagocytosis Journal of Clinical Epigenetics of human polymorphonuclear leukocytes and monocytes or human natural killer cell activity [43] .
    Lastly at the six levels, it was reported that Calcium Channel Blockers (CCBs) reduce the diazepam CIPE in rats and was attributing this anti-inflammatory effect due to stimulation of the HPA axis by CCBs [44] .
    This is consistent with the diazepam report of Schaufele et al . [45] who reported diazepam that diazepam is acting through a common mechanism with calcium antagonists, where some of peripheral and central effects of diazepam are thought to be induced by inhibition of adenosine uptake or by inhibiting calcium channels .
    2: 19 The results of present study with underlying anti-inflammatory mechanisms of diazepam diazepam suggesting that the peripheral benzodiazepine receptor agonists like diazepam could be of high pharmacological interest as a potential anti-inflammatory agent and also a useful tool to increase our understanding of the inflammatory mechanisms and to provide fertile ground for development of new, safe and more effective anti-inflammatory Conflict of Interest The author declares no conflict of interest .

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